Sung-Wook Hong, PhD
Fellowship dates: 2018-2023
Mentor: Marc Jenkins
Fellowship dates: 2018-2023
Mentor: Marc Jenkins
Contact Info: [email protected]
Email: [email protected]
Program: University of Minnesota Medical Student
Year Entered: 2021
Thesis Advisor: Vaiva Vezys (Lab website)
Research: List of publications
Degrees received:
BS, Boston University, 2019
Email: [email protected]
Current:
Fellowship dates: 2023-present
Mentor: Vaiva Vezys
Research Emphasis/Publications: "Understanding mechanisms of peripheral immune tolerance in autoimmunity" Full list of Publications
Research Associate: 2013-2022
Mentor: Marc Jenkins
Research Emphasis/Publications: Group A Strep pathogenesis and Antigen-specific T cell response, pMHCII tetramers, Affinity enhanced pMHCII tetramers. Full list of Publications
Fellowship dates: 2017-2021
Research Emphasis: Focused on memory T cell biology and improving T cell based vaccines.
Current Position: Scientist, Immunitas Therapeutics, Sharon, MA
Contact Info
Program: MICaB
Defense Year: 2022
Advisor: Mike Farrar
Dissertation Title: "Influenza infection recruits distinct waves of regulatory T cells to the lung that limit lung resident IgA+ B cells."
Fellowship dates: 2017-2021
Research Emphasis: Investigating mechanisms controlling generation and maintenance of peripheral T cell tolerance. Researching role of tolerant CD8 T cells in regulating autoimmunity and cancer. Understanding autoimmunity in the context of hygiene hypothesis.
Fellowship dates: 2017-2021
Research Emphasis: The challenge in rheumatoid arthritis is different in that the autoimmune process is driven by peptides that are chemically modified (e.g. citrullinated). In my research, I will use MHC tetramers to identify and characterize pathogenic T-cells in patients with antibody mediated rejection of lung transplant as well as rheumatoid arthritis.
Fellowship dates: 2015-2022
Research Emphasis: Using adoptive cell transfer and p:MHCII-tetramer enrichment with I have demonstrated important mechanisms programing CD4 T helper-1 cells during acute and persistent infections.