Program: Microbiology, Immunology, and Cancer Biology Graduate Student
Year Entered: 2018
Defense Year: 2024
Thesis Advisor: Kristin Hogquist
Dissertation Title: "Characterizing the role of thymic interferon in T cell development and selection"
Research: Negative selection of autoreactive T cells. List of publications
Fellowship dates: 2021-2023
Mentor: Kris Hogquist
Research Emphasis: List of Publications.
Degrees Received:
DO: Rocky Vista University College of Osteopathic Medicine, 2020
MS: University of Colorado Anschutz Medical Campus, 2017
BA: Hendrix College, 2014
Fellowship dates: 2021-2025
Mentor: Kris Hogquist
Research Emphasis: Thymic myeloid populations in mechanisms of central immune tolerance. List of Publications.
Degrees Received:
PhD: Charles University, Czech Republic, 2020
MS: Charles University, Czech Republic, 2014
BS: Charles University, Czech Republic, 2012
Fellowship dates: 2008-2010
Advisor: Kris Hogquist
Research Emphasis: T cells and Thymic development
Research Associate 2010-2016
Laboratory of Dr. Kristin A. Hogquist
Fellowship dates: 2009-2012
Advisor: Kris Hogquist
Fellowship dates: 2004-8
Fellowship dates: 2010-2013
Research Emphasis: Researched the T cell development and modeling of selection events in the thymus.
Current Position: Vice President, Donor Operations, NMDP, Minneapolis, MN
Contact Info: [email protected]
Fellowship dates: 2014-18
Research Emphasis: Investigating function and thymic development of CD8αα intestinal epithelial lymphocytes (IELs). Specifically the two major thymic precursors for CD8αα IELs, which differ in their antigen receptor specificity and developmental cues. Nature Immunology 2017 Jul;18(7):771-779
Fellowship dates: 2002-2006
Research Emphasis: A requirement for sustained ERK signaling during thymocyte positive selection in vivo. and The regulated expression of a diverse set of genes during thymocyte positive selection in vivo.
Fellowship dates: 1997-2002
Advisor: Kris Hogquist
Research Emphasis: The central tolerance response to male antigen in normal mice is deletion and not receptor editing.
